5 More Minutes And Other Lies Your Brain Tells You


By Keerthi Lakshmi Narayanan

~6-minute read


You’ve been studying for over an hour straight. As you get up and stretch lazily, you reach for your phone, for a quick 5-minute break. 1 hour later, you finally surface from a never-ending hole of doomscrolling with no thoughts in your head, woken by your mom calling you down for dinner. Sounds familiar? This is the life of almost every teenager with access to social media.

Social media addiction is easily attributed to “teenage laziness, weak will, or a lack of discipline”, but in reality, these misconceptions obscure the much more complex neuroscience of the pull of social media.

The nucleus accumbens is a structure in your brain that acts like the command center of your brain’s reward system, the mesolimbic dopamine pathway. Its job is relatively simple. When something good happens- you get a new high score on your favourite game, or you get a compliment on your hair- it releases dopamine, a chemical neurotransmitter that labels the experience as “worth repeating again”. 

However, there’s a big problem with this circuit- it evolved long before smartphones existed and thus cannot differentiate a genuinely rewarding experience and a notification ping. Every like, comment, follow request, or streak reminder lights up the same neural pathway that lights up when you win money, eat your favourite snack, and most troublingly, when using drugs and alcohol (Sherman et al., 2025). Researchers at the London South Bank University who study this “dopa-mining” effect directly found that repeated engagement trains the brain to octave those hits, making it progressively harder to resist checking your phone (Kuss, 2025).

Dopamine hits aren’t the only thing shaping our reactions to our phones either. During adolescence, the brain undergoes a process called neural pruning, where it eliminates weaker or less-used synaptic connections while reinforcing and strengthening the ones you exercise the most, similar to how a gardener cuts back stray branches so the strongest ones can grow thicker. Neural pruning is a normal and, in fact, necessary process for our neurological development. However, it is experience-dependent – so whichever circuits are being used the most during these fundamental years of our growth are the ones that survive.

This means that when a teenager spends hours a day chasing quick dopamine hits from the constant buzz of notifications, those reward pathways are consolidated and strengthened while the circuits responsible for sustained attention and delayed gratification are pruned away from disuse (Sherman et al., 2025). 

Things only get harder from here because the part of the brain that’s supposed to apply the brakes isn’t ready yet. The prefrontal cortex, the part of the brain responsible for impulse control and weighing long-term consequences, doesn’t fully mature until the mid-twenties, whilst the amygdala, which processes emotions and social threat, is fully ready in teenage years. This awkward mismatch means that teenagers feel the all-too-strong pull of FOMO, social comparison and the anxiety of being left on delivered far more intensely than adults do, while also not having the ability to resist acting on it.

The effects of social media addiction have far deeper claws into our well-being than we realise. Teens who spend more than three hours a day on social media show measurably impaired impulse control, which closely mirrors the patterns seen in substance-related addictions ( Alam et al., 2025). Researchers describe this escalation in 2 stages- early use is driven by the immediate dopamine hit of likes and validation, but sustained, compulsive use is maintained by a completely different system- a breakdown in the pathways that link the prefrontal cortex to the limbic system leaves people scrolling not because it feels good, but to escape feeling bad (Zhang & Li, 2025). This phenomenon is called digital anhedonia- a diminished capacity to feel pleasure in real-world experiences after prolonged digital overstimulation (Rao, 2025).

None of this means that the only solution is to give up social media entirely, but understanding what goes on in your brain can change what you actually do about it. Turning off non-essential notifications removes a large share of the tiny dopamine hits that pull you back in. Protecting sleep and ensuring that social media does not interfere with your sleep schedule avoids worsening emotional regulation or academic performance. 

At the end of the day, you’re just a teenager whose brain is being expertly reverse-engineered by people who pick apart brains and how to manipulate them for a living- engineers, data scientists, and behavioural psychologists whose entire job is figuring out how to keep you scrolling for one more minute. That’s not a fair fight, and it’s not one you can win through sheer willpower alone. Understanding the science behind it simply puts a weapon in your hands- how you wield it is up to you.


Written by Keerthi Lakshmi Narayanan, published by Viktoriia Dmitrieva.

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Sources: 

Sherman, L. E., et al. (2025). Understanding the brain’s response to social media: A closer look at dopaminergic mechanisms. ReachMD. https://reachmd.com/news/understanding-the-brains-response-to-social-media-a-closer-look-at-dopaminergic-mechanisms/2470999/ 

Kuss, D. (2025, November 19). Social media rewires young minds — here’s how. The Conversation. https://theconversation.com/social-media-rewires-young-minds-heres-how-243120 

Alam, M., et al. (2025). Neurobiological and behavioral correlates of excessive social media use in adolescents. Journal of Surgery and Medicine. https://jsurgmed.com/article/view/8211 

Zhang, Y., & Li, H. (2025). The emotional reinforcement mechanism of and phased intervention strategies for social media addiction. National Center for Biotechnology Information. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC12108933/ 

Rao, V. (2025). Hijacked by the feed: Social media neuroengineering-induced digital anhedonia. National Center for Biotechnology Information. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC12042983/

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